Facial paralysis usually does not cause dizziness, but it may be associated with it in rare cases.

Facial paralysis can lead to facial pain and numbness, and may affect expression and taste. It is recommended to use B vitamins and hormone therapy, and to undergo acupuncture. It is advisable to avoid cold winds to prevent the condition from worsening.

Facial paralysis can sometimes lead to tongue numbness, as the tongue’s movement and sensation may be affected by the condition. In the case of central facial paralysis, abnormal brain nerve function may be one of the causes, which can also affect the tongue’s sensation. It is recommended to undergo a head CT scan to determine if there are vascular diseases and to use relevant medications for treatment.

Even though the symptoms of facial paralysis stabilize after treatment, precautions must be taken to avoid catching a cold or wind, otherwise the disease may recur.

Facial paralysis is not caused by genetics; it is usually due to various factors, including infectious inflammation, otological diseases, and autoimmune reactions.

Facial paralysis does not have a hereditary pattern across generations. Its onset is mainly related to physical factors, although it has a genetic component, it is not gene-linked hereditary. Postnatal adjustment can improve physical condition and prevent the occurrence of the disease.

Facial paralysis may leave sequelae, including abnormal facial expressions, asymmetry of the eyes, and mouth twitches. Early treatment is crucial to reduce the occurrence of sequelae.

Whether facial paralysis has sequelae depends on the severity of the symptoms, the timeliness of treatment, and the system. Mild symptoms treated in the hospital and with appropriate rehabilitation usually do not leave sequelae. Severe symptoms that are not treated promptly may result in sequelae.

Facial paralysis is a relatively complex facial disorder, with most cases caused by facial exposure to cold, physical injury, or viral invasion. Initially, the condition manifests as inflammation of the facial nerve, which is not yet accompanied by evident facial symptoms. As the illness progresses, patients may exhibit typical symptoms such as drooping eyelids and crooked mouth and eyes.

Does facial paralysis lead to muscle atrophy after effects, and how can it be prevented and treated?